In recent years, China's research on imidacloprid seed dressing has never stopped. Many scholars in China are actively exploring different application methods of imidacloprid insecticides on various crops, hoping to find a way to fully exert its systemic activity. It can not only achieve the purpose of poisoning target pests, but also be friendly to non-target organisms and environment, and has a reasonable effect on long-lasting use.
Under the unremitting efforts of many scholars in China, it was finally found in the application of seed dressing that imidacloprid mixed with monocotyledonous plants such as wheat, corn and sorghum can control aphids during the whole growth period; but mixed with dicotyledons such as cotton, aphids, etc. The control effect of sucking mouthparts pests is mostly about 60 days.
The insecticidal mechanism of imidacloprid seed dressing is unique. Imidacloprid is a neurotoxic agent, and its target is the nicotinic acetylcholinesterase receptor of the post-synaptic membrane of the nervous system of the pest, which interferes with the normal stimulation and transmission of the motor nervous system of the pest, and thus manifests as paralysis. lethal. This is different from the conventional insecticides, and thus the insecticide resistant to organophosphorus, carbamate, pyrethroid insecticides, and imidacloprid still have better control effects. It is obvious that it is mixed with or mixed with these three kinds of insecticides.
Although imidacloprid seed dressings have obvious utility, they also have a fatal short board, which is that it is easy to cause pest resistance. Because imidacloprid has a single action site, pests are susceptible to drug resistance, and the number of applications should be strictly controlled during use. In order to avoid the emergence of resistance to pests, continuous use on the same crop is strictly prohibited twice. In 2007 and 2008, the outbreak of rice brown planthopper in the whole country and the continuous use of imidacloprid on rice for many years led to the direct resistance of brown planthopper to imidacloprid and the loss of control effect.
Under the experiment of domestic scholars, it is concluded that imidacloprid can effectively enhance the activity of protective enzymes and root activity in plants at an appropriate concentration, thereby promoting plant growth and improving plant vigor; if the amount of imidacloprid is too large, it will inhibit plant protection. The enzyme system and root activity, which inhibits plant growth, lead to a series of symptoms of phytotoxicity: seed mold, seed germination, growth inhibition, seed setting rate and yield reduction.
From the above we can conclude that the imidacloprid seed dressing effect is obvious, but special care should be taken not to use it frequently. Domestic research on imidacloprid is still in-depth, and it is believed that more applications are being studied.
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